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Mr. Abraham D. Kingford President, CEO King Information Systems 765 Rumson Road Medford, NJ 09876 Dear Mr. Kingford:

resource can change, and the permissions that you apply to that shared content might need to change as well. Also, as new users who should have access to the network share are created, the users must be added to groups that have access to the shared resource.

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The Network Security screen enables the administrator to set parameters that control how the client adapter associates with an AP, authenticates to the wireless network, and encrypts and decrypts data (see Figure 5-8).

There are several specific tasks you can perform on a selected address list. We ll illustrate this using the Marketing Department list we created earlier. Select the All Address Lists node in the Recipients container of System Manager, right-click on the Marketing Department list, and select Properties. The General tab displays in a condensed logical notation the filter you have configured for this list. Click Modify to change your filter or Preview to display the list membership based on the currently configured filter. Remove deletes your current filter if you need to reconfigure the list from scratch. The Security tab lets you manage the permissions associated with the list. We ll cover permissions in more detail in 13, but for now just select Authenticated Users in the top listbox and note that these users have List Contents permission for the address list, enabling them to view the contents of the list. The grayed-out checkbox indicates that this permission is inherited rather than directly applied to the object. Click Advanced and you ll see that the permissions for Authenticated Users are actually more detailed. Other tasks you can perform on address lists using System Manager are renaming a list and deleting it.

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kidneys to excrete a number of substances, specifically phosphate. An almost universal complication of chronic renal failure is elevated plasma phosphate (hyperphosphatemia). Another common finding is elevated levels of PTH. The high PTH stimulates excessive bone resorption, leading to osteoporosis. This is an example of secondary hyperparathyroidism (not primary) because the pathology is not in the gland itself, but in the signals that drive it. One goal in the treatment of hyperphosphatemia associated with chronic renal failure is the reduction of phosphate absorption from the GI tract. This is accomplished by feeding the patient high doses of calcium. The calcium forms complexes with phosphate in the GI tract, reducing the availability of absorbable phosphate. The high levels of PTH in a normal patient should signal the kidneys to form vitamin D, but in chronic renal failure a further complication is a reduced ability to synthesize it. Another clinical intervention in this case is to provide exogenous vitamin D. This hormone suppresses the expression of the PTH gene in the parathyroid gland. The vitamin D should increase GI absorption of phosphate, the very thing we are trying to inhibit, but its ability to lower the synthesis of PTH is more important because this reduces the excessive resorption of bone stimulated by PTH. Thus, administering vitamin D is a useful clinical tool.

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